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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. homemade penis enlarement home penis enargement guide to penis elargement pennis enlargement pic before and after vimax penis enlargement traction device pro solution review easy enlagement free penis surgery way easy enlargement free pnis surgery way

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The Internet is flooded with websites promoting penis enlargement devices, pills, and techniques. Penis enlargement products can range in price from about $30 to over $1000, but are you really getting what you pay for? Pills A product once promoted by a Penthouse Playmate called Longitude proposed that men ingest two of its capsules per day for a larger penis. After one to three weeks, a small increase in penis length and girth was to be expected. During weeks four through eight, the user was to expect a noticeable thickness in the erect and flaccid state. Week nine promised that you will be both thicker and longer (1 inch or more). There was a supposed 30-day money back guarantee for the unsatisfied customer. (I just want to make it clear that this article was written a few years ago and I never tried any of these products. I have updated this article and am bringing this update for continued awareness from an unbiased perspective.) One of the marketing schemes that Longitude used was the fact that all of its ingredients were all natural and completely safe. The ingredients were as follows: Zinc, 300 Yohimbe, Maca, Catuaba, Muira Puama, Oyster Meat, L-Arginine, Oat Straw, Nettle Leaf, Cayenne, Pumpkin Seed, Sarsaparilla, Orchic Substance, Licorice Root, Astragalus, Tribulus, Boron, and Ginseng. After reviewing all of the literature on Longitude's ingredients, I made one conclusion: There was no substantial evidence suggesting that it actually worked. The majority of studies done on each of its ingredients, excluding zinc, arginine, and ginseng, had been conducted on laboratory animals such as rats. In addition, only one or two studies were related to sexual function. It appeared that Longitude worked by stimulating erection, not growth. The company was able to cover this claim easily because the product did what it said it would: increase penis size 1", 2", even 3". Your penis will most likely increase to that size from the flaccid state, especially if stimulated. This was a clever way to market the product because the company never lied to the consumer, only mislead them. Considering that the product took at least 60 days to exert its full effects, the 30-day money back guarantee didn't even make sense. Plus, the fact that you would be monitoring your penis size daily would have most likely lead to results that were not there. In the end, the company was shutdown and penis advertisements on television have become scarce. Penis Pumping Another product marketed to consumers is the Dr. Joel Kaplan vacuum system. The advertisement suggests that it has been FDA approved for penis enlargement. Don't be fooled. The FDA approval is only for its use as an aid to cure erectile dysfunction (not being able to get an erection). This claim has misled consumers thinking that it is proven to increase penis growth. You may experience short-term growth because you are sucking more blood into the penis. However, this may be a risky thing to do. Surgery As of now, the only guaranteed way to increase the size of your penis is through cosmetic surgery, followed by stretching exercises. The downside to this is the risk of damage to the nerves surrounding the area resulting in loss of erectile function; or even worse, an oddly shaped penis. The surgery is quite costly, and is still considered in the experimental stage. Good research has not been conducted in the area of penis enlargement techniques. As of now, there are no supplements that one can take to actually increase penis growth per se. Viagra and Cialis are available for men who lack the ability to achieve and sustain an erection. Most supplements marketed as penis enlargement enhancers use the same principle. The only difference is that they use herbal extracts that have been used in Third World countries for the same thing. The results are not as significant and may pose a threat to health, given the lack of FDA regulation. Aphrodisiacs and stimulants are added to supplements in hopes of increasing libido and producing a harder and firmer erection. Although some products on the market may show short-term benefits in regards to penis enlargement, there is no quick fix to the problem that many men encounter everyday. Research has shown that the average penis size in the flaccid state is 3.9 inches. Erect state averages between five and seven inches. One of the concerns that men have is that the size of their penis in the flaccid state is too small. Showering with other men in the locker room can lead to embarrassment and the feeling of inadequacy. Advertisement of penis enlargement devices will continue to plague the American market. What you must be aware of is the fact that nothing, besides surgery, will permanently increase the size of your penis. Surgery has many dangers associated with it, and the risks far exceed the benefits. Don't waste your money, or risk injuring yourself for something of this magnitude. pennis enlargement pic before and after herbal natural pnis enlargement natural penile enlargment technique vimax penis enlargement review penis enlagement excersizes cheap vigrx pills free pnis enlargement video enlargement forum free matter pnis size home penis enlargment

There are many products that claim to increase libido and keep your penis healthy, however a healthy penis and strong libido are normally a reflection of overall lifestyle. Many men look for miracle cures, when they should be looking at changes in lifestyle. These changes become even more critical as men pass the age of 40. Lets look at 6 simple liefstyle changes that can help you A healthy penis and overall health The human body is a totally integrated organism, it is virtually impossible to have a healthy penis if there are health problems elsewhere. Al healthy penis means, one without erectile dysfunction (ED). ED is not yet perfectly understood, and most studies are inconclusive if the causes are physiological or psychological, or both factors working together. A healthy penis and testosterone levels As men age, it is proved that the level of testosterone in the blood slowly decreases. This is a main cause of libido drop and if libido can effect erections, it is then a cause of ED. Check yourself, and see if any of the following symptoms apply to you. Do you have sleeping problems, hair loss, become tired quickly, gaining weight, depression, become irritable easily, Sweating and flushing without physical activity? So, if your testosterone is dropping, there is no way to have a healthy penis, and you must resort to the new generation of ED drugs (Viagra, Cialis, and so on) which may work, and may not and have unpleasant side effects. Getting an erection with an ED drug does not mean your penis (or you are healthy) Also, if your state of health has deteriorated, you veins and arteries are not open as they were when you were young, so your ED may be caused by a slowdown / breakdown in your circulatory system. A healthy penis 6 rules for increased libido For both levels of testosterone and your general state of health, you can follow the easy rules below. You will be healthier, and it will show with a healthy penis and a more virile you Rules 1, 2 and 3 are MUSTS! 1.If you smoke, stop now. Smoking damages every part of your body, especially the circulatory system, and introduces dangerous chemicals directly into the blood. 2.You need physical exercise, and every day. It is estimated that you need at least 30 minutes of strenuous exercise daily as a minimum. To get testosterone levels up naturally, put yourself on a good weight lifting program. See your doctor first, and then ask a professional trainer to give you a special program suited to your body type, age, and physical condition. The testosterone levels will rise naturally. This is a proven fact. 3.Diet. If you are overweight, your penis will pay the price, along with your heart and every other critical organ. Eat plenty of fruits (in season only), and vegetables, lots of roughage (in the form of cereals), and meat sparingly. Your weight will drop naturally (without a formal diet) and you will notice your libido returning and your ability to satisfy it as well. The next rules are SHOULDS; so keep as much to them as you can. 4.Moderate your alcoholic intake. Do not over-indulge in drinking. Hard liquor sparingly, and wines and beers with great moderation. 5.Find good vitamin and mineral supplements. It should contain most or all of the following: Vitamins A, B, B complex, C, D, and E. From minerals, look for Chromium, Selenium and Zinc. From protein, L-arginine. From enzymes, DHEA. From natural herbs, look for Damania, Dong quai, Ginger, Ginkgo Biloba, Ginseng, Gotu kola, Horny Goat Weed, Hydrangea root, Pygeum, Sarsaparilla, Saw palmetto, and Wild yam. 6.If you are alone, masturbate. You need to ejaculate often, and through masturbation, the chemicals and reactions required for keeping your penis healthy are put to use. Done properly (as in the technique called jelqing) the result is, if nothing more, an improved circulation to the penis, and use of the prostate gland for more than just continence. A healthy penis final words Remember the state of your penis is a good indication of your own state of health. Follow the rules above, and at any age, you will find increased testosterone in the blood, better circulation, relief from many symptoms that may now plague you, and of course, a healthier penis. enlargement manhattan penile vimax penis enlargement tip penis enlarement surgeries natural penis enlargement exercise penis enlargement surgery cost cheap vig rx pill vimax penis enlargement supplement penis elargement surgery picture home penis enlargment

If you want that perfect show dog look in your Shih Tzu you might as well relax to the fact that it involved brushing, brushing, brushing, and daily brushing. A thorough head-to-tail groom is often needed at least every other day if not “daily.” However, Shih Tzu coat textures are very different. You might get by with once weekly brushing if the Shih Tzu’s coat is the type of texture that does not mat and tangle easily. If you have only one Shih Tzu, grooming should not be a problem. It can be quite pleasurable for you and your Shih Tzu. It can be an excellent time to bond with each other, and have nice close little chats with each other. Where do you start? A good place to start grooming your Shih Tzu is on the tummy, the chest and inside the legs…….perhaps……there are other opinions of course, but in this article I will direct you to start with the tummy, the chest and inside the legs. You will need to have your Shih Tzu lie on his side on the table (be sure you have a nice, sturdy table to groom your Shih Tzu). There is nothing worse than a grooming table that wobbles around, except of course a grooming area that is not organized. You also need to have all your equipment organized and ready to use. So now that you have your Shih Tzu lying on its side on the table, with his legs towards you (this can be an accomplishment in itself that may take several short training sessions to get done). If the Shih Tzu just will not cooperate with you, you can start trying to train him by having him lay tummy upwards in your lap with his head facing your chest. Do this exercise first for a few days if he absolutely refuses to lie this way on the grooming table. If the above fails, you might try to groom the chest and tummy while the Shih Tzu sits on the table facing you, holding him up into a begging position by the front paws; this is not easy, since both hands are needed for some parts of the grooming, but you might have to try this as a last resort. If your Shih Tzu mats the tummy will be a prime target for matting. The tummy side of a Shih Tzu is sensitive also, so be gentle in this area. Check where the legs meet the body, this is a prime target area for severe matting as well. Something you don’t want to deal with is severe mats in these areas. These are extra sensitive areas on your Shih Tzu. The best rule to follow is pay close attention to these areas on a daily basis and never allows the mats to occur. The hair around the male penis can also be a very sensitive trouble spot. It is best to keep this area clean and shaved off. Your male Shih Tzu will give you a sigh of relief when you remove the hair in this area. How do you remove knots and mats? If you come across a mat or knot first try to tease it apart with your fingers. Then take a comb and use the end with coarse teeth to tease out the tangle slowly, working from the outside inwards and holding the hair between the knot and the body between your fingers so that you don’t pull too hard and hurt the Shih Tzu. If all else fails, a knot may have to be cut out. Cut into the knotted hair away from the dog several times before gently using the comb to clear away the knot. Never cut across the coat. This will leave an obvious hole. If you will cut down through the length of the coat you will cause much less damage. Note: The hair on the legs is more likely to tangle than anywhere else. So…..pay particular attention to these areas as well to avoid those awful and painful mats. This article continues in Part II of “Life with the Shih Tzu.” This article is FREE to publish with the resource box. penis enlarement tool penis enlargement traction device free penile enlargment exercise natural penis enargement exercise penile enlargment exercise magna rx pill manual penis elargement magna rx patch home penis enlargment

You can throw out most of the management ideas you find in colleges, graduate schools, company training programs, and the like if you’ll do just one, incredibly simple thing: PAY YOUR PEOPLE EXCEPTIONALLY WELL. Management advocates have it backwards, you see. Their pet saying is that the art of management is getting average people to perform exceptionally well. What they leave off is a small tag line. Let me provide you with the entire phrase: “The art of management is getting average people to perform exceptionally well, without paying them anything extra for their productivity.” That’s truly the tacit definition of a good manager, and most of the industrial psychology, job engineering, and yes, management consulting during the past 80 years has been dedicated to this goal. “Make us more money without making us spend more money.” But, alas, human nature and life itself don’t work that way—for long, or without unleashing counter-forces such as unions, restrictive legislation, workers compensation claims, and lawsuits. Somehow, business owners think it’s “cheating” or “dumb” or self-defeating to pay people exceptionally well. They’d prefer to be clever, to invest in labor saving technologies, to cut out the human touch everywhere they can, instead of handing over living, and indeed, prospering wages. But recall, if you will, that practical experiment undertaken by Henry Ford, by no means a soft touch. Ford invented the “$5 Day.” To attract and recruit the very best laborers in America to work on his modern assembly lines he offered that astonishingly high wage. From what I understand, this more than doubled the prevailing rates. People thought Ford was crazy, and I’m sure he was vilified by his fellow captains of industry. But his move paid off. He did get the best available people to work for him, and together, they made very, very good money. From time to time there have been companies that have raised pay substantially, through salaries, profit-sharing, stock options, attractive retirement packages, and the like. And quite often, they’ve reaped a reward from their employees, by way of output and loyalty. But nothing is as direct or as motivating as more money in that pay envelope. Most of the silly stuff you hear about today, such as RESISTANCE TO CHANGE is easily overcome when we align people’s interests and commit to sharing the goodies, all around. Workers that are accused of being change-haters suspect, or actually know, that the “new and improved” work processes that are often mandated by management result in money being removed from their pockets, through downsizing, job enlargement, team-building, and other devices. Are you going to willingly support something that will make you work harder, longer, and for less? “How can we pay people MORE?” should be the question. Answer that, and people will manage themselves and their peers, because they’ll be only too happy to keep a good thing going!